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 Ammonium chloride is a white, fine or coarse, crystalline powder. it is a good fertilizer for important crops in rainy climates, particularly for rice. It is not registered for current use in the U.S., but approved pesticide uses may change periodically and so federal, state and local authorities must be consulted for currently approved uses. Ammonium chloride is used also as a flux in zinc and tin plating; electroplating, electrolytic refining of zinc; etching solutions in manufacture of printed circuit boards; in dry and Leclanche batteries; manufacturing of explosives; flame suppressant; hardener for formaldehyde-based adhesives; mordant for dyes and printing. It is used as medication particularly in diuretics, expectorants. HUMAN EXPOSURE AND TOXICITY: Potential symptoms of overexposure to fumes are irritation of eyes, skin, and respiratory system; cough, dyspnea, pulmonary sensitization. Large doses of ammonium chloride may cause metabolic acidosis secondary to hyperchloremia, especially in patients with impaired renal function. Other adverse effects of excessive ammonium chloride dosage include rash, headache, hyperventilation, bradycardia, progressive drowsiness, mental confusion, and phases of excitement alternating with coma. Calcium-deficient tetany, hyperglycemia, glycosuria, twitching, hyperreflexia, and EEG abnormalities have also been reported. Most of these adverse effects are secondary to ammonia toxicity resulting from inability of the liver to convert the ammonium ion to urea. Because rapid IV injection may increase the likelihood of ammonia toxicity, IV infusions of ammonium chloride should be administered slowly to permit metabolism of ammonium ions by the liver. Patients receiving ammonium chloride should be closely monitored for signs and symptoms of ammonia toxicity such as pallor, sweating, irregular breathing, vomiting, bradycardia, cardiac arrhythmias, local or generalized twitching, asterixis, tonic seizures, and coma. ANIMAL STUDIES: Acute exposure in mice by intravenous administration resulted in hyperventilation and clonic movements which were followed sometimes by tonic extensor convulsions, but usually by profound coma; death was preceded by convulsions, but survivors made complete and rapid recovery. This syndrome was potentiated by short periods of hypoxia. In rabbits, replacement of aq. humor with 1% solution of ammonium chloride has caused considerable hyperemia of iris, but by next day eyes were almost normal, and in another day were completely recovered. The ingestion of ammonium chloride in doses of around 500-1000 mg/kg bw/day, for periods ranging from 1 to 8 days, has induced metabolic acidosis in mice, guinea-pigs, rats, rabbits, and dog. Pulmonary edema, central nervous system dysfunction, and renal changes are reported to have occurred after ingestion of ammonium chloride